Download Clinical Endocrinology of Dogs and Cats: An Illustrated by Ad Rijnberk, Hans S. Kooistra PDF

By Ad Rijnberk, Hans S. Kooistra

This hugely illustrated textbook is aimed toward the veterinarian in perform or education attracted to every kind of endocrine illnesses of canine and cats. The publication is organised through physique approach and is case oriented.

Chapters take care of separate endocrine glands, starting with a short overview of the morphology and body structure of the gland, whole with drawings depicting the useful rules. this is often by means of descriptions of the problems of the gland with updated details on prognosis and treatment.

Numerous medical circumstances illustrate the offering signs and the consequences following remedy. the ultimate chapters comprise diagnostic and healing protocols, in addition to algorithms for difficulties resembling alopecia, polyuria, and weight-loss.

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Additional resources for Clinical Endocrinology of Dogs and Cats: An Illustrated Text, Second, Revised and Extended Edition

Sample text

A) A healthy beagle. 0 mm). (B) A twelve-year-old female mongrel greyhound with pituitary-dependent hypercortisolism. 2 mm). (C) A ten-year-old female Australian terrier with dexamethasone-resistant pituitary-dependent hypercortisolism without noticeable neurological symptoms. 7 mm). (D) A 14-year-old castrated male domestic shorthair cat presented with mild symptoms and signs of pituitary-dependent hypercortisolism and central blindness. 9 mm). but it is cumbersome to perform them separately.

27) and MRI (fig. 28: Sagittal MR images of the skulls of a healthy dog (A) and a dog with pituitary-dependent hypercortisolism (B). In the healthy dog the hypophyseal cleft between the anterior lobe and the neurointermediate lobe can be distinguished (arrow). In the dog with pituitary-dependent hypercortisolism there is suprasellar extension of the pituitary mass. 155 Dynamic helical CT and MRI also allow visualization of the posterior lobe. 156 Treatment Anterior pituitary failure can be treated by substitution of the hormones inadequately produced by the target glands.

They are rather large, single-chain polypeptides containing 190 (GH) and 199 (PRL) amino acids and having two (GH) or three (PRL) intrachain disulfide bridges (fig. 3). 1 Pituitary growth hormone GH release is characterized by rhythmic pulses and intervening troughs (fig. 10). The GH pulses predominantly reflect the pulsatile delivery of GHRH from the hypothalamus, whereas GH levels between pulses are primarily under the control of somatostatin (SS) or somatotropin-release inhibiting factor (SRIF) (fig.

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